In inflamed skin, such as that in atopic dermatitis, recent evidence shows a shift from type‐2 innate lymphoid cells (ILC2) towards type‐3 innate lymphoid cells (ILC3) that can produce the chemo‐attractive cytokines interleukin‐17 (IL‐17) and IL‐26, thus amplifying the immune reaction and thus engendering inflammation [17]. The gene discussed is IL26; the disease is atopic eczema.