LPA and coronary artery disorder: Lp(a) is considered proatherogenic, proinflammatory, and prothrombotic, with cumulative data suggesting a causal relationship with atherosclerotic cardiovascular disease (ASCVD) [7]; CAD risk has been associated with both high Lp(a) molar concentration and apo(a) size, which are inversely correlated properties of the Lp(a) particle that is mostly genetically determined and highly heterogeneous in the general population [8].