Researchers have discovered that hydrogen intervention in sepsis models, specifically CLP mice and LPS-induced BV-2 cells, significantly inhibits the phosphorylation level of mTOR (indicated by a decrease in the p-mTOR/mTOR ratio) while activating the AMPK signaling pathway (indicated by an increase in the p-AMPK/AMPK ratio). Here, MTOR is linked to Sepsis.