Besides its direct effect on cancer suppression, BCP can interact selectively with the body’s CB2 receptor activation by inhibiting the production of pro-inflammatory cytokines, helping to create an anti-tumor immune environment that suppresses pro-inflammatory cytokines (e.g., IL-6, TNF-α) and promotes anti-inflammatory mediators to inhibit tumor initiation and progression [28]. This evidence concerns the gene IL6 and neoplasm.