However, the additional functionality of identification of this histological phenotype of lung cancer is not just at a genomic level but perhaps more effectively at transcriptomic levels, emphasizing that cell proliferation pathways (example: E2F, G2M, and MYC), upregulated in SCLC and large-cell neuroendocrine cancer as compared to LUAD [127]. Here, MYC is linked to small cell lung carcinoma.