Aryl hydrocarbon receptor (AhR) activation suppresses autophagy and promotes skin inflammation through the NF-Kappa B (NF-κB)/p38 mitogen-activated protein kinase (MAPK) signaling pathway [20], leading to inflammatory cytokine secretion (IL-1β, IL-6, and TNF-α) in keratinocytes [18]. This evidence concerns the gene NFKB1 and dermatitis.