This role is further strengthened by previous demonstration of C15:0 as a means to prevent and treat ferroptosis and tissue iron deposition; its demonstrated activities as an antioxidant, anti-inflammatory, and pro-mitochondrial activities, as well as being an mTOR inhibitor (which supports autophagy) and AKT activator, all of which have been shown to support cognitive health and proposed as potential approaches to treating AD in humans [2,50,65,66,67,68,69]. The gene discussed is AKT1; the disease is Alzheimer disease.