In addition to C15:0’s demonstrated ability to reverse all components of ferroptosis, a promising neuroprotective target to prevent and treat AD, this study also showed that C15:0 has dose-dependent FAAH and MAO-B inhibition activities, both of which have neuroprotective and cognitive health-supporting mechanisms that also have the potential to prevent and attenuate AD in humans. Here, MAOB is linked to Alzheimer disease.