The parallels in microglial activation, altered expression of inflammation-related genes, and enrichment of proinflammatory signaling pathways between Chm-cKO choroideremia mice and Mertk-related RP models [23] suggest a strong similarity in the retinal inflammation-related features at the molecular and cellular levels in choroideremia and RP. This evidence concerns the gene MERTK and retinitis pigmentosa 1.