Harakeh et al. [67] demonstrated that wadi and talh honey increased SOD and CAT expression in indomethacin-induced gastric ulcer models, while Malkoç et al. [78] observed that rhododendron honey enhanced wound healing in diabetic rats by upregulating GPx, SOD, and CAT expression and reducing the TNF-α levels. The gene discussed is SOD1; the disease is gastric ulcer.