DMD and Duchenne muscular dystrophy: Another case corresponds to the use of adenine bases for the correction of Duchenne muscular dystrophy (DMD) to modify the splice donor sites of the dystrophin gene, causing skipping of a common DMD deletion mutation of exon 51 by the associated adenovirus serotype 9 that modifies adenine bases, restoring dystrophin expression in human pluripotent stem cell-derived cardiomyocytes.