In line with these results, Deniz and colleagues found in FH patients with subclinical atherosclerosis, that Lp(a) levels ≥ 0.3 g/L predicted coronary atherosclerosis, while diabetes and low Apo A‐I/Apo B ratios predicted carotid atherosclerosis, and smoking predicted femoral atherosclerosis [32]. Here, APOA1 is linked to familial hyperaldosteronism.