For example, in post-myeloproliferative neoplasm secondary acute myeloid leukemia the mutation in JAK2 increases nuclear \documentclass[12pt]{minimal}\usepackage{amsmath}\usepackage{wasysym}\usepackage{amsfonts}\usepackage{amssymb}\usepackage{amsbsy}\usepackage{mathrsfs}\usepackage{upgreek}\setlength{\oddsidemargin}{-69pt}\begin{document}$$\beta$$\end{document}β catenin levels and its co-localization with TBL1, promoting growth and survival [32]. This evidence concerns the gene JAK2 and myeloproliferative disorder.