Indeed, single‐cell RNA sequencing of the alveolar compartment in COVID‐19 patients supports the centrality of AMs in the immune circuits that drive pulmonary inflammation in severe disease [3, 4]—SARS‐CoV‐2 infected AMs have increased expression of cytokines and chemokines including IL‐1β, CCL2,and CXCL10 and recruit memory T cells to the lung. The gene discussed is IL1B; the disease is COVID-19.