LKB1-deficient cell lines with high ΔAUC values exhibited robust apoptosis upon combined inhibition of KRAS/MAPK and MCL-1, while the apoptotic response of LKB1 wild-type (WT) cell lines was minimal (Fig. 1E, F), suggesting that LKB1 may modulate apoptotic dependencies of KRAS-mutant lung cancers. Here, MCL1 is linked to lung carcinoma.