The gene expression module correlating to high tumoral BCR diversity prominently featured the hallmark genes of T cell activation such as CD69 (R = 0.42, FDR < 0.001), IFNG (R = 0.37, FDR < 0.001), and TNF (R = 0.29, FDR < 0.001), reinforcing the potential role of activated CD8+ T cells in mediating the effects of increased BCR diversity within the tumor microenvironment. Here, CD69 is linked to neoplasm.