Similarly, male SLE non-HTN and SLE-HTN subjects exhibited significantly higher anti-ETAR autoantibody levels relative to non-SLE HTN (p = 0.0015 and p = 0.0268, respectively) and non-SLE non-HTN groups (p = 0.0027 and p = 0.0255; Fig. 2Eand Supplemental Table 12). The gene discussed is EDNRA; the disease is hypertensive disorder.