Moreover, consistent with our findings in HG/F-induced H9C2 myoblasts, H&E staining, TUNEL assays, BAX and caspase 3/9 Western blotting, and Masson’s trichrome staining showed that treatment with A-MG alleviated myocardial damage, cardiomyocyte apoptosis, and myocardial fibrosis supplementation in DCM mice. Here, BAX is linked to familial dilated cardiomyopathy.