In this study, to explore a potential cross-linking between oxidative stress and inflammation in FRDA, we investigated the expression of GLRX1, TRX1, NF-kB, and IL-1β in fibroblasts of three patients with FRDA, in order to highlight whether the redox imbalance might activate some inflammatory response in patients. Here, NFKB1 is linked to Friedreich ataxia.