We then established stable CRABP2 overexpression and short‐hairpin (sh)RNA‐mediated CRABP2‐knockdown cell lines (Figure S1H,I, Supporting Information) and used these cells to establish subcutaneous tumors in BALB/c nude mice.[14] Results show that CRABP2 overexpression enhanced tumor growth relative to control cells, leading to increased levels of Ki‐67 staining and decreased levels of TUNEL staining (Figure 1H), with CRABP2 knockdown having the opposite effect (Figure 1I). This evidence concerns the gene MKI67 and neoplasm.