initially identified a maternal oxytocin‐mediated excitatory‐to‐inhibitory GABA polarity switch during parturition, which serves to protect the neonatal brain during delivery.[14] A blunted GABA polarity switch during delivery has been linked to autism‐related pathologies.[15, 33] Importantly, we detected a peak in the driving force of GABA rebound to a high level at P2 in the control rats after parturition. Here, OXT is linked to autism.