Extensive research has identified molecular mechanisms related to inflammation in the kidney caused by diabetes, including the activation of oxidative stress (Charlton et al., 2020), Toll-like receptors (Rayego-Mateos et al., 2020), Janus kinase/signal transducer and also known as protein kinase B or PKB (AKT)/mammalian target of rapamycin (AKT-mTOR) signaling pathways, as well as the activation of nuclear factor kB (NF-kB) (Sanz et al., 2010). This evidence concerns the gene AKT1 and diabetes mellitus.