To assess whether KDM2A expression and atherosclerosis were consistently sharing the true casual variant instead of confounding of linkage disequilibrium, KDM2A was supported by suggestive evidence (PPH4 > 50%) through colocalization analysis, indicating promising probability for a shared casual variant between gene expression and atherosclerosis risk (Figure 10C and Supplementary table 24). Here, KDM2A is linked to atherosclerosis.