Based on the previous results obtained by Korkmaz et al. [14], including experimental pneumonia in LOX-1 KO mice and anti-LOX-1 antibodies, and the long-standing description of neutrophil lung sequestration during ARDS [36], we may speculate that neutrophils and macrophages were captured in the lung parenchyma where they expressed LOX-1 to promote M2-like macrophage polarization and tissue repair in postoperative ARDS patients. This evidence concerns the gene OLR1 and susceptibility to pneumonia measurement.