However, reduced brain weight and delayed gyrification on fetal MRI in CHD53 and reduced doublecortin+ neuroblasts, cortical growth, and NeuN/calretinin+ interneurons, but not NeuN/Tbr1+ neurons, in models of perinatal ischemia, support an additional developmental neuronal‐migratory defect.54 This evidence concerns the gene CALB2 and ischemia.