NFKB1 and metabolic dysfunction-associated steatotic liver disease: [44] demonstrated that H2 altered the composition of the GM, leading to an increase in the relative abundance of Mycobacterium anisopliae and Mycobacterium thickum, while concurrently inhibiting the LPS/TLR 4/NF‐κB inflammatory pathway, thereby preventing HFD‐induced NAFLD.