CDKN2A and glioblastoma: Specifically, we examined: (a) normal human skin fibroblasts BJ induced to drug‐induced senescence (DIS) by docetaxel (DTX); (b) a senescent‐like phenotype in RPE‐1 cells induced by doxycycline (DOX)‐regulated ectopic expression of CDK protein inhibitors p21waf1 (p21) or p16INK4A (p16; [25, 26]); and (c) a senescent‐like phenotype in two glioblastoma cell lines, U‐87 MG and A‐172, induced by the anticancer drug temozolomide (TMZ).