CEBPB and liver disorder: We found that improved fibrosis resolution in KO mice was alcohol specific, because we did not observe C/EBPβ-mediated fibrosis resolution in a metabolic dysfunction–associated steatotic liver disease model, and it was associated with changes in cholesterol/bile acid metabolism and increased expression of the CYP3A family of enzymes, suggesting that these pathways might be involved in fibrosis resolution.