YTHDF2 knockdown could also rescue the decrease in CSF1 mRNA stability under FTO knockdown conditions, confirming that YTHDF2 contributes to CSF1 m6A modification, even though we did not observe differences in its expression between hybrids and corresponding GBM cells (Fig. 7I; Supplementary Fig. S6G). This evidence concerns the gene FTO and glioblastoma.