In addition, in patients with Alzheimer’s disease, LF supplementation led to the decreased expression of phosphatase and tensin homolog (PTEN), tau, and mitogen-activated protein kinase (MAPK1), as well as decreased serum levels of Aβ42, which may indicate its beneficial effects on oxidative stress and inflammatory processes [152]. Here, PTEN is linked to early-onset autosomal dominant Alzheimer disease.