Notably, LLPS in the skeletal muscle has rarely been reported, with factors such as FUS and TDP‐43 being linked to amyotrophic lateral sclerosis (ALS), a neurodegenerative disease affecting muscle function.[52, 53] Our findings suggest that similar LLPS mechanisms may also be at play in muscle cells, with a directly impact on muscle health and disease. This evidence concerns the gene FUS and neurodegenerative disease.