Additionally, non‐survivors exhibited increased concentrations of multiple biomarkers involved in endothelial dysfunction (angiopoietin 2, endothelin‐1, ICAM‐1 and VCAM‐1), inflammation (TNF‐α, IL‐15 and IL‐6), coagulation (D‐dimmer), chemotaxis (CXCL10, CCL2, and IL‐8), immunosuppression (IL‐10, PD‐L1, and IL1‐RA), T‐cell biology (CD27), apoptosis (Fas) and innate immune‐related proteins (EGF and SP‐D). The gene discussed is IL15; the disease is endothelial dysfunction.