We also observed that vaginal infection with GV results in elevated levels of TNF-α, IL-6, and RANKL, as well as an elevated number of NF-κB-positive cells in the vagina, femur, hippocampus, and hypothalamus, leading to vaginitis, osteitis, and neuroinflammation, as previously reported [22]. The gene discussed is NFKB1; the disease is infective vaginitis.