In U2OS cells, TSA promoted autophagy by inhibiting the mTOR (mammalian target of rapamycin) signaling pathway and enhancing forkhead box O1 (FOXO1) transcriptional activity [141], while it induced autophagy through regulating the PRMT5/STC1/TRPV6/JNK pathway in cervical cancer cells [142]. This evidence concerns the gene MTOR and cervical carcinoma.