The current network pharmacology-based analysis of A. laxiflora suggested that compounds (quercetin, 3-acetylursolic acid, and 3-acetyloleanolic acid) in this plant can influence key cancer-associated targets EGFR, AKT1, SRC, and MAPK1 through the PI3K-Akt, MAPK, Ras, and Rap1 signaling pathways. The gene discussed is EGFR; the disease is cancer.