miR-520a is also downregulated in AML and is recognized as a tumour suppressor, according to studies by Chen et al. and Xiao et al. The first author correlated reversely the expressions of miR-520a-3p and MUC1, demonstrating that overexpression of miR-520a-3p inhibited proliferation and induced apoptosis of AML cells (THP-1) [149]. This evidence concerns the gene MUC1 and acute myeloid leukemia.