The results revealed that compared with the control group, the apoptotic proteins p-CSF-1R/CSF-1R, p-Nrf2/Nrf2, and Bcl-2/Bax, associated with neuronal injury and degenerative diseases, were significantly decreased, and cleaved caspase-3 expression was significantly increased. The gene discussed is CASP3; the disease is neurodegenerative disease.