Tumor initiation and progression in cancers of the breast, pancreas, prostate, lung, and intestine and other tissues are driven by several factors such as mutations in oncogenes like KRAS (KRASG12D), loss of tumor suppressors such as p53, adenomatous polyposis coli (APC), and the PTEN lipid and protein phosphatase [4,5,6,7]. Here, APC is linked to neoplasm.