Research has revealed that the development of glycerol-induced AKI is a complex process involving multiple mechanisms, including myoglobin toxicity (Gburek et al., 2003; Reeder and Wilson, 2005), inflammation (Al Asmari et al., 2017), reactive oxygen species (ROS) production (Kim et al., 2010; Wu et al., 2017), and apoptosis (Homsi et al., 2010). The gene discussed is MB; the disease is acute kidney injury.