Previous reports addressing the pathogenesis of uveitis have implicated T-helper type 1 (Th1) and Th17 lymphocytes and their predominantly produced cytokines, interferon-gamma (IFN-γ) and interleukin (IL)-17, respectively, as the cause of exacerbated inflammation associated with uveitis (3, 16). This evidence concerns the gene IL17A and uveitis.