NFKB1 and metabolic dysfunction-associated steatotic liver disease: ROS induces nuclear factor kappa light chain enhancer of activated B cells (NF-κB) activation, disrupting redox signaling, and leading to apoptosis (via caspase 3), plasma membrane rupture (ferroptosis), and further ROS generation, all of which contribute to NAFLD pathophysiology [10–12], through ferroptosis reaction on lipids.