We observed only two significant pathways associated with the commonly upregulated genes, i.e., “Cell cycle” and “Oocyte meiosis.” In the case of MI, upregulated DEGs were majorly enriched for “Lysosome,” “Osteoclast differentiation,” “NOD-like receptor signaling pathway,” “TNF signaling pathway,” etc. Similarly, upregulated genes in LUAD and LUSC show enrichment of common pathways such as “cell cycle,” “Oocyte meiosis,” and the “Fanconi anemia pathway,” as well as unique pathways such as “Motor proteins” in LUAD and “p53 signaling pathway,” “Retinol metabolism,” etc. in LUSC. The gene discussed is TNF; the disease is Fanconi anemia.