The NaBC1 boron (B) transporter is encoded by the SLC4A11 gene and is a Na+‐coupled B co‐transporter that controls B homeostasis.[22] Mutations in the SLC4A11 gene are involved in rare diseases, such as endothelial corneal dystrophies.[23] Previous studies have reported a role for B in osteogenic differentiation[24] and adipogenesis inhibition[25]; however, B function and homeostasis are not completely understood. The gene discussed is SLC4A11; the disease is Fuchs endothelial corneal dystrophy.