Another relevant finding of our study was that SSTR1 and SSTR2 expression in GBM-tissues (dominant SSTR-subtypes expressed in GBM-samples and consistently downregulated across the 5 human analysed cohorts), were associated with malignant features (i.e., OS/recurrence/IDH1-status/GCIMP-status/molecular Verhaak-subtypes/EGFR-mutations). The gene discussed is EGFR; the disease is glioblastoma.