BA was reported to significantly reduce the infarct area and myocardial enzymes (CK, CK-MB, LDH, and cTnT) and inhibit the activity and protein expression of Caspase-3, which was related to the mitogen-activated protein kinases (MAPK) cascade in acute myocardial infarction in rats (Liu et al., 2013). The gene discussed is TNNT2; the disease is myocardial infarction.