Interestingly, mice that have a myeloid-specific lack of Mir26b also show increased hepatic cholesterol levels and lipid accumulation demonstrated by Oil-red-O staining, coinciding with an increased hepatic Cd36 expression (Figure 2), demonstrating that myeloid Mir26b plays a major, but not exclusive, role in the observed steatosis. The gene discussed is CD36; the disease is steatosis.