Our results revealed that, compared to para-cancerous tissues, the expression levels of SLC14A1, NEFH, MSMB, KRT23, and KRT15 were significantly downregulated in PCa tumor tissues, while ARHGEF38 expression was notably upregulated, consistent with our initial predictions (Figures 9A–F). This evidence concerns the gene SLC14A1 and posterior cortical atrophy.