Acute promyelocytic leukaemia (APL) is a specific type of acute myeloid leukaemia (AML) characterised by t(15;17) translocation, promyelocytic leukaemia gene‐retinoic acid receptor alpha (PML::RARA) fusion oncoprotein transcripts and abnormal promyelocyte predomination (1, 2). The gene discussed is RARA; the disease is acute promyelocytic leukemia.