In our flow cytometry results, the positive correlation between CD16+ (FCGR3A+) monocytes and disease activity supports the notion that these cells contribute to the pathogenesis of AAV, as reported in other inflammatory diseases such as rheumatoid arthritis, coronary artery disease, and type 2 diabetes mellitus [18, 19]. The gene discussed is FCGR3A; the disease is type 2 diabetes mellitus.