Inhibition of FATP4 and CD36 in BCCs and TME leads to dysregulation of FA transport and intracellular FA transport in TME; FATP4 and CD36 also participate in cancer cell apoptosis through other mechanisms such as SGLT2 (sodium–glucose cotransporter 2) cotransporter and AMPK (AMP-activated protein kinase)/mTOR (mechanistic target of rapamycin kinase) pathway.14 This evidence concerns the gene SLC27A4 and cancer.