Since KCNJ13 mutations have not been linked with nanophthalmia, rather leading to more severe retinal disorders (Leber congenital amaurosis and snowflake vitreoretinal degeneration), the potential role of the MFRP–KCNJ13 interaction in ocular development and homeostasis remains unresolved and also requires further investigation. The gene discussed is KCNJ13; the disease is Leber congenital amaurosis.